Fig. 7: The knockdown of YBX1 or G3BP1 partially rescues the tumor-promoting effects caused by RECQL4 overexpression in LUAD cells. | Cell Death Discovery

Fig. 7: The knockdown of YBX1 or G3BP1 partially rescues the tumor-promoting effects caused by RECQL4 overexpression in LUAD cells.

From: RECQL4 promotes the malignant progression of lung adenocarcinoma through the YBX1/G3BP1-mediated NF-κB signaling pathway

Fig. 7: The knockdown of YBX1 or G3BP1 partially rescues the tumor-promoting effects caused by RECQL4 overexpression in LUAD cells.The alternative text for this image may have been generated using AI.

A Colony formation assays, B CCK-8 assays, C EdU incorporation assays, D flow cytometry analyses, E wound-healing assays, and F Transwell assays for the rescue experiments in different transfected groups. G Western blotting assays of NF-κB signaling pathways and cell-cycle-related markers were performed in the rescue experiments using A549 cells. *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001; RECQL4 RecQ like helicase 4, YBX1 Y box binding protein 1, G3BP1 GTPase-activating protein SH3 domain-binding protein 1, LUAD lung adenocarcinoma, CCK-8 cell counting kit 8, EdU 5-ethynyl-2’-deoxyuridine.

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