Fig. 1

BA-TGR5-GRK-β-arrestin-SRC axis promotes antiviral immune response. Immediate-early activation of NF-κB induced by virus infection promotes intracellular accumulation of BAs. BAs then activate the TGR5-GRK-β-arrestin-SRC signaling and facilitate activation of multiple innate antiviral signaling components, which finally enhance antiviral immune responses