Extended Data Fig. 9: The cpk3/4/5/6/11 quintuple mutant exhibited shoot growth defects and elevated levels of defense marker gene expression under hyperosmotic stress.
From: TIR immune signalling is blocked by phosphorylation to maintain plant growth

a,b, Shoot growth of Col-0, cpk3-2 mutant (a), and cpk3/4/5/6/11 quintuple mutant (b), 15 days post-transfer to 1/2 MS medium containing 0 mM or 100 mM mannitol. Scale bars, 0.5 cm. c,d, Levels of relative expression of PR1 (c) and PR2 (d) in Col-0 and cpk3/4/5/6/11 quintuple mutant five days after transfer to the 1/2 MS medium containing 0 or 100 mM mannitol. All experiments were repeated at least three times with similar results. Error bars represent means ± SD (n = 3). Asterisks denote significant differences (two-way ANOVA with Tukey’s test, ****P < 0.0001).