Table 1 Demographic characteristics of individuals referred with a clinical diagnosis of iRBD with vs. without DAT binding deficit

From: Basic clinical features do not predict dopamine transporter binding in idiopathic REM behavior disorder

 

DAT binding deficit present (n = 49)

No DAT binding deficit (n = 26)

p-value for differences between groups

Mean age in years (SD; Min; Max)

69.84 (6.2; 51.0, 84.7)

69.38 (7.7; 60.0, 86.3)

0.7774

Male: Female n (%)

40 (81.63):9 (18.37)

19 (73.08):7 (26.92)

0.3894

Education

   

<13 Years n (%)

20 (40.82)

15 (57.69)

0.1633

 ≥ 13 Years n (%)

29 (59.18)

11 (42.31)

 

Family history of PD

  

0.8981

Yes n (%): No n (%)

7 (14.29):42 (85.71)

4 (15.38):22 (84.62)

 

Handedness right n (%): Left or mixed n (%)

44 (89.80):5 (10.20)

24(92.31):2 (7.69)

0.7219

Mean RBD disease duration from diagnosis in years (SD; Min; Max)b

2.85 (3.1; 0.1–11.8)

3.58 (3.4; 0–9.9)

0.3615

Mean RBD disease duration from symptom onset in years (SD; Min; Max)b

9.61 (6.8; 0.38–30.26)

3.58 (3.4; 0.03–9.90)

0.6484

REM sleep without atonia on polysomnogram confirmed by the PPMI sleep corea, n (%)

41 (97.62)

20 (95.24)

0.6114

  1. aTwelveparticipants did not have polysomnographic data available for review by the sleep core (in these cases, polysomnogram had been performed and showed REM sleep without atonia, but the raw polysomnographic data were not available for tranfer to the central sleep core for confirmation). Seven of these cases were in the DAT Deficit+ group and 5 in the DAT Deficit− group
  2. bRBD disease and symptom duration was missing on two subjects
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