Extended Data Fig. 4: Maz loss in MNs engenders features of CTCF-boundary disruption. | Nature Genetics

Extended Data Fig. 4: Maz loss in MNs engenders features of CTCF-boundary disruption.

From: CRISPR and biochemical screens identify MAZ as a cofactor in CTCF-mediated insulation at Hox clusters

Extended Data Fig. 4

a, GO analysis of biological processes in differentially expressed genes in MAZ KO ESCs compared to WT ESCs. PANTHER overrepresentation test tools were used (see Methods) and GO processes with a fold enrichment > 2 were presented (FDR < 0.05). b, GO analysis of biological processes in differentially expressed genes in MAZ KO MNs compared to WT MNs. PANTHER overrepresentation test tools were used (see Methods) and GO processes with a fold enrichment > 2 were presented (FDR < 0.05). c, RNA-seq MA plots of WT versus MAZ KO ESCs at the HoxA, HoxC, and HoxD clusters. d, RNA-seq MA plots of WT versus MAZ KO MNs at the HoxA, HoxC, and HoxD clusters. RNA-seq results represent three biological replicates. Hox genes with RNA abundance ≥ 10 are represented in colors, and the rest are represented in gray (see Supplementary Data 10-11). Hox genes in 3 Hox clusters are colored based on their position with respect to the previously demonstrated CTCF-boundary in MNs. e, RT-qPCR analysis for the indicated Hox genes in HoxA cluster, HoxC cluster (f), and HoxD cluster (g) in WT, MAZ KO, and CTCF(Δ5|6:6|7) cells. RT-qPCR signal is normalized to Gapdh levels. Fold change in expression in MNs is calculated relative to baseline expression in ESCs. All RT-qPCR results are represented as mean values and error bars indicating SD across three biological replicates. Maz KO represents three independent clones. Supplementary Data 12 shows raw data for RT-qPCR and the comparison for each gene in WT versus MAZ KO and WT versus CTCF(Δ5|6:6|7) in both ESCs and MNs.

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