Table 3 Latency to white spot (WS) formation and first hatchling emergence between treatments.

From: Synchronised nesting aggregations are associated with enhanced capacity for extended embryonic arrest in olive ridley sea turtles

 

Nesting tactic

Control

3-day

3.5-day

4-day

8-day

15-day

30-day

Mean latency to WS (d)

Non-arribada

0.8 ± 0.0A*

3.5 ± 0.1B

—

4.8 ± 0.1C

8.8 ± 0.1D

16.0 ± 0 E

—

Arribada

1.0 ± 0.0X*

3.4 ± 0.2Y

—

4.6 ± 0.1Z

—

—

—

Mean aerobic latency to WS (d)

Non-arribada

0.8 ± 0.4A*

0.5 ± 0.1A

—

0.8 ± 0.1A

0.8 ± 0.1A

1.0 ± 0A

—

Arribada

1.0 ± 0.0X*

0.4 ± 0.2Y

—

0.6 ± 0.1Y

—

—

—

Latency to first hatchling emergence (d)

Non-arribada

49

52

53

52

—

—

—

Arribada

50

52

51

51

54

57

—

  1. Olive ridley eggs were collected from non-arribada and arribada nesting females and incubated following different durations of post-oviposition hypoxia. The number of eggs from each treatment group that formed a white spot after incubation in normoxia are shown in Table 2. All eggs from the 3.5-day and 30-day treatment formed WSs while in hypoxia, as did the 8- and 15-day arribada eggs, so there are no data for latency to white spot. Furthermore, no 30-day eggs hatched, nor did any 8- and 15-day non-arribada eggs, so there are no data for latency to hatchling emergence. When superscript letters are the same, there was no significant between-group difference within nesting tactic according to an ANOVA and Tukey’s HSD post-hoc test. An asterisk (*) denotes a significant difference within each treatment between each nesting tactic according to an ANOVA and Tukey’s HSD post-hoc test.
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