Figure 1 | Scientific Reports

Figure 1

From: Dysfunctional EAT thickness may promote maladaptive heart remodeling in CVD patients through the ST2-IL33 system, directly related to EPAC protein expression

Figure 1

EAT thickness directly correlates with ST2 expression and inversely with IL-33. (a) End-diastolic (a1-a2) and end-systolic (b1-b2) echocardiographic frames, showing EAT thickness (red arrows). (b) Correlation results among EAT measurement and both ST2 molecular expression (r = 0.54, p < 0.0001) and IL-33 (Spearmann r = −0.50; p < 0.01) suggesting a potential involvement of fat body increase in ST2/IL-33 regulation.

Back to article page