Table 6 Correlation pattern of EAT biopsies from CVDs patients with highets sST2 (>57.42 pg/ml).

From: Dysfunctional EAT thickness may promote maladaptive heart remodeling in CVD patients through the ST2-IL33 system, directly related to EPAC protein expression

Correlation pattern of EAT biopsies from CVDs patients with highets sST2 (>57,42 pg/ml)

X

Y

Spearman r

p

IL1RL1 (ST2) gene expression

EPAC 2 (AU)

0.73

0.007

sST2 total circulating level

NT-pro BNP (pg/ml)

0.65

0.002

HbA1 (%)

0.37

0.04

IL-33 expression gene (AU)

0.7

0.008

IL-33 gene expression

sST2 total circulating levels (pg/ml)

0.69

0.008

IL1RL1 expression gene (AU)

−0.51

0.05

EPAC2 gene expression

ST2 expression gene (AU)

0.66

0.01

  1. Correlation patterns of CVD patients expressing higher sST2 total circulating levels than the median of the total CVD population (57.42 pg/mL). CVD population releasing high levels of sST2 protein into the circulation presented a correlation pattern associated with maladaptive remodeling. Spearman correlation analysis was used for relations among different variables.
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