Figure 6

Schematic of the mechanism by which involvement of cav-1 and caveolae in CM-induced beta cell apoptosis in pancreatic beta-cells. Cytokine mixture treatment into beta cells inhibited insulin secretion and induced apoptosis. Cytokine mixture treatment increased caveolae structure as well as cav-1 expression and cytokine receptors (TNFR1 and IL1-R1) were recruited into caveolae. Therefore, activation of NF-kB signaling pathway increased the expression level of inflammatory response genes, which leads to beta cell apoptosis.