Table 3 The effect of CME against diniconazole genotoxicity.

From: Preventive efficiency of Cornelian cherry (Cornus mas L.) fruit extract in diniconazole fungicide-treated Allium cepa L. roots

Damage

Group 1

Group 2

Group 3

Group 4

Group 5

Group 6

MN

0.56 ± 0.64d

0.42 ± 0.54d

0.24 ± 0.38d

35.70 ± 5.13a

27.80 ± 4.85b

19.50 ± 3.92c

MI

754.60 ± 25.94a

765.30 ± 26.82a

776.40 ± 27.66a

430.50 ± 19.48d

490.60 ± 21.64c

550.20 ± 22.85b

FRG

0.00 ± 0.00d

0.00 ± 0.00d

0.00 ± 0.00d

60.80 ± 7.52a

50.30 ± 6.84b

38.20 ± 5.65c

SC

0.36 ± 0.48d

0.26 ± 0.34d

0.12 ± 0.26d

40.40 ± 4.86a

32.50 ± 4.34b

24.90 ± 3.76c

UDC

0.00 ± 0.00d

0.00 ± 0.00d

0.00 ± 0.00d

30.40 ± 3.74a

22.60 ± 3.26b

15.80 ± 2.88c

VN

0.00 ± 0.00d

0.00 ± 0.00d

0.00 ± 0.00d

21.40 ± 2.36a

15.80 ± 1.96b

9.40 ± 1.18c

  1. *Data are shown as mean ± SD (n = 10). Group 1: control, Group 2: 0.5 g/L CME, Group 3: 1.0 g/L CME, Group 4: 100 mg/L diniconazole, Group 5: 0.5 g/L CME + 100 mg/L diniconazole, Group 6: 1.0 g/L CME + 100 mg/L diniconazole. For MN and CAs 1,000 cells were counted in each group and 10,000 cells for MI. MN micronucleus, MI mitotic index, FRG fragments, SC sticky chromosome, UDC unequal distribution of chromatin, B bridge, VN vacuole nucleus. The averages shown with different letters (a–d) in the same line are statistically significant (p < 0.05).