Figure 2
From: An apocrine mechanism delivers a fully immunocompetent exocrine secretion
SG phenotype in eygC1/eygC53 mutants. (a) Longitudinal SEM view of the wild-type (Oregon R) SG at the end of the 3rd instar, with (b) detailed perpendicular view of its opening into the SG duct. (c) Laser confocal image of the anterior end of a wild-type 3rd larval instar SG showing a few columnar cells and the duct [tumor suppressor protein p127l(2)gl (green), nuclear transcription factor BR–C (red), and filamentous actin (blue)]. (d) SEM view of a eygC1/eygC53 mutant salivary gland, with (e) a detailed perpendicular view of the mutilated, closed anterior end of the gland lacking a duct. (f) Optical section of the anterior end of eygC1/eygC53 mutant 3rd larval instar SG showing that it is a closed sack having a few columnar cells and is missing a duct (channels as above).
