Table 2 Demographic and clinical features of the cohorts (n = 157).

From: Automated in-depth cerebral arterial labelling using cerebrovascular vasculature reframing and deep neural networks

Cases

Control (n = 42)

Stroke with ICAS

(n = 46)

Stroke (n = 69)

p-value*

p-value†

p-value‡

Sex, male

31 (73.8)

27 (58.7)

77 (67.5)

0.138

 < 0.001

0.433

Age, years

58 ± 10.1

64.2 ± 13.3

69.3 ± 12.8

0.018

 < 0.001

 < 0.001

Height, cm

166.3 ± 8.3

161.5 ± 9.7

163.4 ± 9

0.016

0.923

 < 0.001

Weight, kg

67.4 ± 10.9

63.7 ± 12.5

65.8 ± 11.4

0.148

0.637

0.008

Hypertension

16 (38.1)

29 (63)

74 (66.7)

0.019

0.01

 < 0.001

Diabetes

5 (11.9)

18 (42.9)

31 (27.9)

0.003

0.877

0.003

Hyperlipidaemia

11 (26.2)

18 (42.9)

56 (50.5)

0.201

0.006

 < 0.001

aCurrent smoking

6 (16.7)

11 (23.9)

14 (20.3)

0.671

0.067

0.162

NIHSS

 

3.2 ± 4.2

2.8 ± 3.4

 

0.524

 

TOAST40,

    

0.459

 

Large artery atherosclerosis

 

34 (73.9)

12 (17.4)

  

Cardioembolism

 

0 (0.0)

16 (23.2)

  

Small vessel occlusion

 

0 (0.0)

16 (23.2)

  

Other determined etiology

 

1 (2.2)

3 (4.3)

  

Undetermined etiology

 

6 (13.0)

10 (14.5)

  
  1. aThe intensity of smoking ranges from zero to two.
  2. *Boldened if p < 0.05. P-values statisticalanalysedsed by homoscedastic two-sample t-test had two-tailed distribution parameters between the controls and stroke-with-ICAS patients.
  3. †Homoscedastic two-sample t-test had two-tailed distribution parameters between the stroke-with-ICAS and stroke-only patients.
  4. ‡Homoscedastic two-sample t-test had two-tailed distribution parameters between the controls and stroke-only patients.
  5. AF, atrial fibrillation; TOAST, Trial of Org 10,172 in Acute Stroke Treatment.
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