Table 1 Clinical characteristics in patients with type 2 MI.

From: Management and outcome trends in type 2 myocardial infarction: an investigation from the SWEDEHEART registry

Demographics

Type 2 MI (n = 14,883)

Type 1 MI (n = 182,879)

 

Missing data

 

Missing data

Age (years)

78 (69–85)

72 (63–80)

Men

7494 (50.4%)

122,773 (67.1%)

Risk factors

 Current smoking

1859 (12.5%)

10

35,191 (19.2%)

53

 Hypertension

9523 (64.0%)

8

104,346 (57.1%)

90

 Diabetes

4080 (27.4%)

11

40,282 (22.0%)

131

 Hyperlipidemia

6631 (44.6%)

18

62,622 (34.3%)

257

 Body mass index (kg/m2)

25.8 (23.1–29.2)

1393

26.6 (24.1–29.7)

10,191

 eGFR (mL/min/1.73m2)

61.5 (41.2–82.0)

630

76.3 (56.8–90.1)

5489

Comorbidities

 Previous MI

5563 (37.4%)

13

51,391 (28.1%)

118

 Previous PCI/CABG

4082 (27.5%)

13

44,286 (24.2%)

121

 Heart failure

2754 (18.5%)

13

18,105 (9.9%)

131

 Atrial fibrillation (adm.)

3777 (25.4%)

40

16,626 (9.1%)

538

 Previous stroke

1904 (12.8%)

14

14,736 (8.1%)

144

 Peripheral artery disease

1752 (11.8%)

11,654 (6.4%)

 COPD

2538 (17.1%)

13,942 (7.6%)

 Previous/present cancer

1247 (8.4%)

8229 (4.5%)

Diagnostic procedures

 Echocardiography

9735 (65.4%)

149,386 (81.9%)

 Coronary assessment

5301 (39.6%)

89,681 (79.5%)

Discharge medications*

 Betablockers

10,814 (78.3%)

144,893 (85.9%)

 RAAS–inhibitors

5990 (75.6%)

95,643 (87.3%)

 Statins

9296 (66.1%)

158,811 (91.1%)

Discharge medications in patients with CAD*

 Betablockers

6183 (85.5%)

not assessed

 RAAS–inhibitors

3539 (78.0%)

not assessed

 Statins

5632 (76.5%)

not assessed

  1. *Data from hospital survivors (all type 2 MI: n = 14,116; type 2 MI with CAD: n = 7391; type 1 MI: n = 174,866).
  2. Data are given as numbers with percentages, and as medians with 25th and 75th percentiles.
  3. MI Myocardial infarction; eGFR Estimated glomerular filtration rate; PCI Percutaneous coronary intervention; CABG Coronary artery bypass grafting; adm: admission; COPD Chronic obstructive pulmonary disease; RAAS Renin–angiotensin–aldosterone-system; CAD coronary artery disease.