Fig. 7
TAMs retain proinflammatory plasticity in the TME. Dynamic, reciprocal interaction between tumor-polarized macrophages and activated T cells allows paracrine reprogramming of TAM via IFNG secretion.
Fig. 7 | Scientific Reports
TAMs retain proinflammatory plasticity in the TME. Dynamic, reciprocal interaction between tumor-polarized macrophages and activated T cells allows paracrine reprogramming of TAM via IFNG secretion.
