Table 3 GO and KEGG enrichment analysis results of OXSRDEGs.

From: Oxidative stress gene expression in ulcerative colitis: implications for colon cancer biomarker discovery

Ontology

ID

Description

p.adjust

qvalue

BP

GO:0,030,593

Neutrophil chemotaxis

0.001327

0.000755

BP

GO:0,090,594

Inflammatory response to wounding

0.067463

0.03839

BP

GO:1,903,209

Positive regulation of oxidative stress-induced cell death

0.069035

0.039285

BP

GO:0,035,883

Enteroendocrine cell differentiation

0.074564

0.042431

BP

GO:0,050,900

Leukocyte migration

0.001889

0.001075

CC

GO:0,034,774

Secretory granule lumen

0.082046

0.046061

CC

GO:0,060,205

Cytoplasmic vesicle lumen

0.082046

0.046061

CC

GO:0,031,983

Vesicle lumen

0.082046

0.046061

CC

GO:0,062,023

Collagen-containing extracellular matrix

0.086317

0.048458

CC

GO:0,035,580

Specific granule lumen

0.086317

0.048458

MF

GO:0,005,125

cytokine activity

5.62E−06

3.15E−06

MF

GO:0,008,009

chemokine activity

6.01E−05

3.36E−05

MF

GO:0,048,018

receptor ligand activity

8.86E−05

4.96E−05

MF

GO:0,030,546

signaling receptor activator activity

8.86E−05

4.96E−05

MF

GO:0,001,664

G protein-coupled receptor binding

0.00252

0.001411

KEGG

hsa04657

IL-17 signaling pathway

1.84E−05

9.97E−06

KEGG

hsa04060

Cytokine-cytokine receptor interaction

0.000688

0.000373

KEGG

hsa04668

TNF signaling pathway

0.000736

0.000399

KEGG

hsa04062

Chemokine signaling pathway

0.002711

0.00147

KEGG

hsa04064

NF-kappa B signaling pathway

0.011772

0.006384

  1. GO gene ontology, BP biological process, CC cellular component, MF molecular function, OXSRDEGs oxidative stress related differentially expressed genes.