Fig. 8: Schematic illustration of CEACAM6-mediated EGFR signaling in lung adenocarcinoma.

CEACAM6 interacts with EGFR, enhancing EGFR stability and maintaining epithelial morphology, thereby promoting lung cancer cell growth. TGF-β stimulation and epigenetic regulation downregulate CEACAM6, shifting toward EGFR-independent signaling and consequent EGFR-TKI resistance characterized by elevated EMT features.