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Showing 1–7 of 7 results
Advanced filters: Author: Ørjan Samuelsen Clear advanced filters

  • Plasmids can encode multiple traits that contribute to the emergence and dissemination of their bacterial hosts. Here the authors use Nanopore long-read technology to sequence the complete genomes of <2,000 bloodstream infection E. coli isolates, highlighting the contribution of chromosomal and plasmid-encoded traits in the success of different lineages.

    • Sergio Arredondo-Alonso
    • Anna K. Pöntinen
    • Kyriakos Zaragkoulias
    ResearchOpen Access
    Nature Communications
    Volume: 16, P: 1-15

  • E coli is an important cause of infection globally but there is limited understanding of the genetic diversity of strains circulating in South Asia. Here, the authors characterise strains in Punjab province, Pakistan by assembling >5,000 genomes sampled from outpatients and community dwellers.

    • Tamim Khawaja
    • Tommi Mäklin
    • Anu Kantele
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-11

  • Opportunistic bacterial pathogen species frequently colonise the human gut as a normal part of the ecosystem but strain-level colonisation and competition dynamics in healthy hosts is yet to be established. Authors seek to understand the relationship between colonisation potential and ecological factors modulating pathogen prevalence in disease in a longitudinal cohort.

    • Tommi Mäklin
    • Harry A. Thorpe
    • Jukka Corander
    ResearchOpen Access
    Nature Communications
    Volume: 13, P: 1-13

  • Little is known about the distribution, evolution and functions of the K1 capsule at a population level, despite the important role in the pathogenesis of E. coli; authors explore this through the utilisation of over 5,000 clinical isolates in population genomics studies and statistical modelling.

    • Sergio Arredondo-Alonso
    • George Blundell-Hunter
    • Alex J. McCarthy
    ResearchOpen Access
    Nature Communications
    Volume: 14, P: 1-17

  • Resistance to one antibiotic can in some cases increase susceptibility to other antibiotics. Here, Podnecky et al. study these collateral responses in E. coli clinical isolates and show that efflux-related resistance mechanisms and relative fitness of the strains are principal contributors to this phenomenon.

    • Nicole L. Podnecky
    • Elizabeth G. A. Fredheim
    • Pål J. Johnsen
    ResearchOpen Access
    Nature Communications
    Volume: 9, P: 1-11