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Showing 1–5 of 5 results
Advanced filters: Author: Alessandro Camponeschi Clear advanced filters

  • Men develop larger myocardial infarction (MI) sizes than women. Here, the authors show that male sex and testosterone, via bone marrow stroma, exacerbates MI-induced neutrophilia and cardiac injury and that response to anti-inflammatory treatment in MI is greater in men than women

    • Elin Svedlund Eriksson
    • Marta Lantero Rodriguez
    • Åsa Tivesten
    ResearchOpen Access
    Nature Communications
    Volume: 16, P: 1-16

  • X-chromosome inactivation (XCI) contributes to sex bias in T cell immunity, but data on profiling XCI during human T cell development is still lacking. Here, the authors leverage allele-specific expression, sex-biased gene expression and DNA methylation data on human pediatric thymocytes to find surprisingly stable XCI during thymocyte differentiation.

    • Björn Gylemo
    • Maike Bensberg
    • Colm E. Nestor
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-14

  • Self-reactive B cells producing autoantibodies are associated with autoimmune conditions. Here, the authors show that in mice lacking the surrogate light chain, which normally assembles with antibody heavy chain to form a pre-B-cell receptor, the autoantibody-producing cells derive from germinal centres.

    • Ola Grimsholm
    • Weicheng Ren
    • Inga-Lill Mårtensson
    Research
    Nature Communications
    Volume: 6, P: 1-12

  • Lipid nanoparticles (LNPs) are potential platforms for RNA-based therapeutics, but the fate of LNP-RNAs upon internalization into the cell is unclear. Here, the authors show that LNP-mRNAs and ionizable lipids escape the endosomes and are re-released via extracellular vesicles which could deliver the functional mRNA to other cells.

    • Marco Maugeri
    • Muhammad Nawaz
    • Hadi Valadi
    ResearchOpen Access
    Nature Communications
    Volume: 10, P: 1-15

  • Testosterone deficiency is associated with autoimmunity and increased B cell numbers, but the underlying mechanism is unclear. Here the authors show that testosterone may modulate the production of B cell survival factor BAFF by fibroblastic reticular cells via regulation of splenic neurotransmitter levels.

    • Anna S. Wilhelmson
    • Marta Lantero Rodriguez
    • Åsa Tivesten
    ResearchOpen Access
    Nature Communications
    Volume: 9, P: 1-13