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Showing 1–5 of 5 results
Advanced filters: Author: Andreas M. Fritzen Clear advanced filters

  • Here, Miranda-Cervantes et al. identified pantothenate kinase 4 (PanK4) as a key regulator of muscle metabolism. Deleting PanK4 impairs fatty acid oxidation and glucose uptake, leading to glucose intolerance, while increasing PanK4 enhances glucose metabolism, highlighting its potential in promoting metabolic health.

    • Adriana Miranda-Cervantes
    • Andreas M. Fritzen
    • Maximilian Kleinert
    ResearchOpen Access
    Nature Communications
    Volume: 16, P: 1-15

  • Muscle mass is lost in patients with diabetes, which is associated with mitochondrial disfunction. Here they show that SLIRP maintains muscle mitochondria and that exercise training can compensate for SLIRP loss, improving mitochondrial function and quality control in muscle.

    • Tang Cam Phung Pham
    • Steffen Henning Raun
    • Lykke Sylow
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-21

  • The physiological role of GDF15 remains poorly defined. Here, the authors show that circulating GDF15 increases in response to prolonged exercise, but that this exercise-induced GDF15, unlike pharmacological GDF15, does not affect post-exercise food intake or exercise motivation.

    • Anders B. Klein
    • Trine S. Nicolaisen
    • Christoffer Clemmensen
    ResearchOpen Access
    Nature Communications
    Volume: 12, P: 1-9

  • This Review summarizes the adaptations to lipid metabolism that occur in skeletal muscle in response to either a high-fat diet or exercise training. In addition, obesity-associated impairments in fatty acid oxidation capacity and the importance of exercise for overcoming lipid metabolic inflexibility in obesity are discussed.

    • Andreas Mæchel Fritzen
    • Anne-Marie Lundsgaard
    • Bente Kiens
    Reviews
    Nature Reviews Endocrinology
    Volume: 16, P: 683-696