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Advanced filters: Author: Brandon E. Mauch Clear advanced filters

  • Williams et al. report a growth arrest mechanism in residual cancer persister cells through targeted therapy-induced upregulation of type I interferon signalling, which is negatively regulated by apoptotic DNA endonuclease DFFB to allow tumour relapse.

    • August F. Williams
    • David A. G. Gervasio
    • Matthew J. Hangauer
    ResearchOpen Access
    Nature Cell Biology
    Volume: 27, P: 2143-2151