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A reversible SRC-relayed COX2 inflammatory program drives resistance to BRAF and EGFR inhibition in BRAF^V600E colorectal tumors

Coppé and colleagues define a resistance mechanism to combination BRAF- and EGFR-targeted therapy in BRAF^V600E colorectal cancers that could be overcome in vitro and in vivo by repurposing COX2 inhibitors.

Ana Ruiz-Saenz
Chloe E. Atreya
Jean-Philippe Coppé
ResearchOpen Access09 Feb 2023
Nature Cancer

Volume: 4, P: 240-256

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A reversible SRC-relayed COX2 inflammatory program drives resistance to BRAF and EGFR inhibition in BRAFV600E colorectal tumors

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A reversible SRC-relayed COX2 inflammatory program drives resistance to BRAF and EGFR inhibition in BRAF^V600E colorectal tumors