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Correction: Corrigendum: Regulation of monocyte cell fate by blood vessels mediated by Notch signalling

Jaba Gamrekelashvili
Roberto Giagnorio
Florian P. Limbourg
Amendments and CorrectionsOpen Access03 May 2017
Nature Communications

Volume: 8, P: 1
The ARNT–STAT3 axis regulates the differentiation of intestinal intraepithelial TCRαβ⁺CD8αα⁺ cells

Intraepithelial lymphocytes (IELs) defend against pathogens in the gut, and their differentiation depends on both diet and microbiota-derived factors. Nakajima et al. show that signalling through the aryl hydrocarbon receptor nuclear translocator and STAT3 is required for the development of a subset of IELs.

Kohei Nakajima
Yoichi Maekawa
Koji Yasutomo
Research09 Jul 2013
Nature Communications

Volume: 4, P: 1-11
Notch2 integrates signaling by the transcription factors RBP-J and CREB1 to promote T cell cytotoxicity

How T cell cytotoxic activity is induced remains incompletely defined. Yasutomo and colleagues now show that Notch2 signals, in direct cooperation with the transcription factor CREB1, promote granzyme B expression.

Yoichi Maekawa
Yoshiaki Minato
Koji Yasutomo
Research24 Aug 2008
Nature Immunology

Volume: 9, P: 1140-1147
Regulation of monocyte cell fate by blood vessels mediated by Notch signalling

Circulating Ly6C^lo monocytes are thought to be derived from Ly6C^hi subset. Here the authors show that Notch signalling is activated in Ly6C^locells and is required for their differentiation, and that Notch ligands that initiate this signalling are provided by a subset of endothelial cells.

Jaba Gamrekelashvili
Roberto Giagnorio
Florian P. Limbourg
ResearchOpen Access31 Aug 2016
Nature Communications

Volume: 7, P: 1-15
Notch controls the survival of memory CD4⁺ T cells by regulating glucose uptake

Yoichi Maekawa
Chieko Ishifune
Koji Yasutomo
Research15 Dec 2014
Nature Medicine

Volume: 21, P: 55-61
Transmission of survival signals through Delta-like 1 on activated CD4⁺ T cells

Takahiro Furukawa
Chieko Ishifune
Koji Yasutomo
ResearchOpen Access23 Sept 2016
Scientific Reports

Volume: 6, P: 1-11
MicroRNA-449a deficiency promotes colon carcinogenesis

Masanori Niki
Kohei Nakajima
Koji Yasutomo
ResearchOpen Access06 Sept 2017
Scientific Reports

Volume: 7, P: 1-10

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Correction: Corrigendum: Regulation of monocyte cell fate by blood vessels mediated by Notch signalling

The ARNT–STAT3 axis regulates the differentiation of intestinal intraepithelial TCRαβ+CD8αα+ cells

Notch2 integrates signaling by the transcription factors RBP-J and CREB1 to promote T cell cytotoxicity

Regulation of monocyte cell fate by blood vessels mediated by Notch signalling

Notch controls the survival of memory CD4+ T cells by regulating glucose uptake

Transmission of survival signals through Delta-like 1 on activated CD4+ T cells

MicroRNA-449a deficiency promotes colon carcinogenesis

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Quick links

The ARNT–STAT3 axis regulates the differentiation of intestinal intraepithelial TCRαβ⁺CD8αα⁺ cells

Notch controls the survival of memory CD4⁺ T cells by regulating glucose uptake

Transmission of survival signals through Delta-like 1 on activated CD4⁺ T cells