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Showing 1–2 of 2 results
Advanced filters: Author: Joel Hrit Clear advanced filters

  • The transmission of unrepaired DNA lesions through mitosis needs tight control. Here, the authors uncover a damaged chromatin marking mechanism driving the segregation of UV damage through mitosis with potential consequences on daughter cell fate.

    • Juliette Ferrand
    • Juliette Dabin
    • Sophie E. Polo
    ResearchOpen Access
    Nature Communications
    Volume: 16, P: 1-12

  • The histone variant H3.3 is phosphorylated at Ser31 in induced genes, and this selective mark stimulates the histone methyltransferase SETD2 and ejects the ZMYND11 repressor, thus revealing a role for histone phosphorylation in amplifying de novo transcription.

    • Anja Armache
    • Shuang Yang
    • Steven Z. Josefowicz
    Research
    Nature
    Volume: 583, P: 852-857