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Showing 1–6 of 6 results
Advanced filters: Author: Jordi Gouilly Clear advanced filters

  • Decidual natural killer (NK) cells from the pregnant uterus play an important role in the physiology of pregnancy and differ functionally from peripheral blood NK cells. Siewiera et al. reveal that this is partly due to the differential expression of splice variants of natural cytotoxicity receptors by these two cell subsets.

    • Johan Siewiera
    • Jordi Gouilly
    • Nabila Jabrane-Ferrat
    ResearchOpen Access
    Nature Communications
    Volume: 6, P: 1-12

  • Herpes simplex virus 1 (HSV-1) is a neurotropic virus that often cause pain via the induction of ulcer or blisters. Here the authors show, in mouse models of HSV-1 infection, that the pain-perceiving nociceptor Nav1.8+ neurons contribute to regulating both innate and adaptive immune responses against HSV-1, thereby offering a potential target for therapy.

    • Jessica Filtjens
    • Anais Roger
    • Sophie Ugolini
    ResearchOpen Access
    Nature Communications
    Volume: 12, P: 1-15

  • Zika virus (ZIKV) infection of pregnant women is associated with pregnancy loss and birth defects, but molecular insights for the aetiology are scarce. Here the authors show that ZIKV reprograms the host lipidome to facilitate viral replication, induce mitochondria dysfunction, and cause immune imbalance, thereby identifying a potential target for ZIKV therapy.

    • Qian Chen
    • Jordi Gouilly
    • Nabila Jabrane-Ferrat
    ResearchOpen Access
    Nature Communications
    Volume: 11, P: 1-16

  • Specialized somatosensory neurons that express Gαi-interacting protein (GINIP) promote tissue repair in a model of UV-induced skin damage by secreting the neuropeptide TAFA4, which regulates skin-resident macrophages to release IL-10 and limit fibrosis.

    • Guillaume Hoeffel
    • Guilhaume Debroas
    • Sophie Ugolini
    Research
    Nature
    Volume: 594, P: 94-99

  • Hepatitis E virus (HEV) infection can result in severe placental disease, but mechanisms underlying pathogenicity are poorly understood. Here, the authors develop an ex vivo model for HEV infection at the maternal-fetal interface and compare pathogenicity of different HEV genotypes.

    • Jordi Gouilly
    • Qian Chen
    • Hicham El Costa
    ResearchOpen Access
    Nature Communications
    Volume: 9, P: 1-13