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Showing 1–11 of 11 results
Advanced filters: Author: Junqi Kuang Clear advanced filters

  • Emerging evidence suggests that exit from pluripotency is a regulated, rather than passive process. Here the authors identify a requirement for SS18-mediated Brg/Brahma-associated factors (BAF) chromatin remodeling complex assembly during exit from pluripotency, and that SS18 promotes BAF assembly through liquidliquid phase separation.

    • Junqi Kuang
    • Ziwei Zhai
    • Duanqing Pei
    ResearchOpen Access
    Nature Communications
    Volume: 12, P: 1-11

  • Cell fate is principally regulated by a common machine or cell-intrinsic machine. Here the authors report the design and testing of an engineered chromatin controller to drive such machine for mouse cell fate transition in precision.

    • Tao Huang
    • Dong Liu
    • Duanqing Pei
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-11

  • Cell fate decisions remain poorly understood at the molecular level. Here Ming et al. show that BMP4 as the signal diverting cell fate away from epiblast/pluripotency to hypoblast/primitive endoderm fate during JGES reprogramming by promoting the dissociation of SALL4 from NuRD.

    • Jin Ming
    • Lihui Lin
    • Duanqing Pei
    ResearchOpen Access
    Nature Communications
    Volume: 15, P: 1-13

  • Somatic reprogramming involves both transcriptional and epigenetic resetting, but we don’t yet fully understand this process. Here they show that Jdp2, Glis1, Esrrb, and Sall4 can mediate reprogramming by recruiting the NuRD complex to close chromatin, highlighting a potential role in cell fate control.

    • Bo Wang
    • Chen Li
    • Duanqing Pei
    ResearchOpen Access
    Nature Communications
    Volume: 14, P: 1-14

  • Volumetric additive manufacturing of protein scaffolds has a wide range of possible biomedical applications. Here the authors report on the bioprinting of unmodified silk sericin and silk fibroin inks with shape-memory and tuneable mechanical properties and demonstrate the potential of the inks in different applications.

    • Maobin Xie
    • Liming Lian
    • Yu Shrike Zhang
    ResearchOpen Access
    Nature Communications
    Volume: 14, P: 1-17

  • Pei and colleagues report that the oncogene c-Jun acts as a somatic cell reprogramming barrier, and that its truncated dominant-negative form can substitute for Oct4 in reprogramming.

    • Jing Liu
    • Qingkai Han
    • Duanqing Pei
    Research
    Nature Cell Biology
    Volume: 17, P: 856-867