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Showing 1–6 of 6 results
Advanced filters: Author: Katja Gehmlich Clear advanced filters

  • Filamin C is a key actin-binding protein involved in cardiomyopathies and musculoskeletal disorders. Here, Wang et al reveal that it interacts with the heat shock protein HSPB7 under biomechanical stress, forming a stable hetero-dimer which is regulated by phosphorylation.

    • Zihao Wang
    • Guodong Cao
    • Justin L. P. Benesch
    ResearchOpen Access
    Nature Communications
    Volume: 16, P: 1-16

  • Most scientific prizes and medals are named after men, and most of these are also awarded to men. The very few awards named after women or not named after a person at all are more frequently awarded to women, although parity between the gender of recipients is still not achieved. We call on the scientific community to rethink the naming of academic awards, medals and prizes, their nomination and selection criteria, and to diversify awarding committees and procedures to ensure greater inclusivity.

    • Katja Gehmlich
    • Stefan Krause
    Comments & Opinion
    Nature Human Behaviour
    Volume: 8, P: 190-193

  • Altered function of the muscle LIM protein (MLP) causes dilated cardiomyopathy in mice and humans. Lange et al. explain the molecular role of MLP in the heart by showing that it affects the signalling complex at the intercalated discs of failing hearts that consists of PKCα, PLCβ1 and CARP by inhibiting PKCα auto-phosphorylation and function.

    • Stephan Lange
    • Katja Gehmlich
    • Elisabeth Ehler
    ResearchOpen Access
    Nature Communications
    Volume: 7, P: 1-11

  • AMPK regulates cellular energy balance using its γ subunit as an energy sensor of cellular AMP and ADP to ATP ratios. Here, the authors show that γ2 AMPK activation lowers heart rate by reducing the activity of pacemaker cells, whereas loss of γ2 AMPK increases heart rate and prevents the adaptive bradycardia of endurance training in mice.

    • Arash Yavari
    • Mohamed Bellahcene
    • Houman Ashrafian
    ResearchOpen Access
    Nature Communications
    Volume: 8, P: 1-19