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Mucosal Immunology (2)
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Advanced filters: Author: Natasha Jaiswal Clear advanced filters

GRK specificity and Gβγ dependency determines the potential of a GPCR for arrestin-biased agonism

Using cytosolic or membrane-tethered GRK2/3 mutants and G protein inhibitors, authors show that G protein activation, releasing free Gβγ, and GRK2/3-mediated β-arrestin recruitment are inseparably intertwined. This adds to the concept of biased agonism.

Edda S. F. Matthees
Jenny C. Filor
Carsten Hoffmann
ResearchOpen Access03 Jul 2024
Communications Biology

Volume: 7, P: 1-12
Correction: Airway epithelial cells prime plasmacytoid dendritic cells to respond to pathogens via secretion of growth factors

Farah Rahmatpanah
Sudhanshu Agrawal
Anshu Agrawal
Amendments and Corrections31 Jan 2019
Mucosal Immunology

Volume: 12, P: 841
Airway epithelial cells prime plasmacytoid dendritic cells to respond to pathogens via secretion of growth factors

Farah Rahmatpanah
Sudhanshu Agrawal
Anshu Agrawal
Research02 Oct 2018
Mucosal Immunology

Volume: 12, P: 77-84

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GRK specificity and Gβγ dependency determines the potential of a GPCR for arrestin-biased agonism

Correction: Airway epithelial cells prime plasmacytoid dendritic cells to respond to pathogens via secretion of growth factors

Airway epithelial cells prime plasmacytoid dendritic cells to respond to pathogens via secretion of growth factors

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