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Advanced filters: Author: Patrick Hannan Clear advanced filters

    • PATRICK J. HANNAN
    • JOHN F. CHRISTMAN
    • RUSTUM ROY
    Correspondence
    Nature
    Volume: 317, P: 106

  • In this study, Aggarwal and colleagues perform prospective sequencing of SARS-CoV-2 isolates derived from asymptomatic student screening and symptomatic testing of students and staff at the University of Cambridge. They identify important factors that contributed to within university transmission and onward spread into the wider community.

    • Dinesh Aggarwal
    • Ben Warne
    • Ian G. Goodfellow
    ResearchOpen Access
    Nature Communications
    Volume: 13, P: 1-16

  • Techniques for tagging active neurons with high spatiotemporal precision are limited. Here the authors report soma-targeted CalLight (ST-Cal-Light) which selectively converts somatic calcium rise triggered by action potentials into gene expression, and generate a conditional ST-Cal-Light knock-in mouse.

    • Jung Ho Hyun
    • Kenichiro Nagahama
    • Hyung-Bae Kwon
    ResearchOpen Access
    Nature Communications
    Volume: 13, P: 1-16

  • The kākāpō is an intensively managed parrot endemic to New Zealand. Using genome sequencing data for all living kākāpō together with long-term phenotypic data, the authors devise an approach to identify genetic associations with fitness traits, which is informing species recovery plans.

    • Joseph Guhlin
    • Marissa F. Le Lec
    • Peter K. Dearden
    Research
    Nature Ecology & Evolution
    Volume: 7, P: 1693-1705

  • Aimee Deaton et al. identify a rare missense variant in the bile acid receptor gene NR1H4, which is associated with lower levels of total cholesterol in the Icelandic population. Hepatocytes expressing the missense variant showed altered expression of a small number of genes, with enrichment in lipid-related pathways.

    • Aimee M. Deaton
    • Patrick Sulem
    • Kari Stefansson
    ResearchOpen Access
    Communications Biology
    Volume: 1, P: 1-9

  • Rajesh Thakker and colleagues show that missense mutations affecting codon 15 of AP2S1 cause familial hypocalciuric hypercalcemia type 3, a disorder of calcium homeostasis. AP2S1 encodes a protein involved in clathrin-mediated endocytosis, and the mutations probably cause disease by disrupting internalization of the calcium-sensing receptor CaSR.

    • M Andrew Nesbit
    • Fadil M Hannan
    • Rajesh V Thakker
    Research
    Nature Genetics
    Volume: 45, P: 93-97