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Publisher Correction: miR-379 deletion ameliorates features of diabetic kidney disease by enhancing adaptive mitophagy via FIS1

Mitsuo Kato
Maryam Abdollahi
Rama Natarajan
Amendments and CorrectionsOpen Access04 Feb 2021
Communications Biology

Volume: 4, P: 1-2
Enrichment of the exocytosis protein STX4 in skeletal muscle remediates peripheral insulin resistance and alters mitochondrial dynamics via Drp1

STX4 is a regulator of the glucose transporter GLUT4 vesicle fusion at the plasma membrane in skeletal muscle. Here, the authors show that skeletal muscle-specific STX4 enrichment reverses established insulin resistance and improves mitochondrial function in the context of diabetogenic stress.

Karla E. Merz
Jinhee Hwang
Debbie C. Thurmond
ResearchOpen Access20 Jan 2022
Nature Communications

Volume: 13, P: 1-14
miR-379 deletion ameliorates features of diabetic kidney disease by enhancing adaptive mitophagy via FIS1

Kato, Abdollahi et al. identify a redox protein thioredoxin and mitochondrial fission-1 (FIS1) protein as miR-379 targets in mouse kidney. They find that miR-379 knockout mice are protected from diabetic kidney disease by enhancing mitophagy via FIS1, suggesting miR-379 as a potential therapeutic target for diabetic kidney disease.

Mitsuo Kato
Maryam Abdollahi
Rama Natarajan
ResearchOpen Access04 Jan 2021
Communications Biology

Volume: 4, P: 1-17

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