Null GRIN2A human patients display a largely transient seizure burden that resolves with age, which may be attributable to a transient delay in the developmental maturation of parvalbumin-positive interneurons in CA1 as is observed in Grin2a+/− and Grin2a-/- mice.
- Chad R. Camp
- Anna Vlachos
- Stephen F. Traynelis
