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Showing 1–4 of 4 results
Advanced filters: Author: S. J. Matkovich Clear advanced filters

  • Myeloid cells are involved in hypertension, but their exact role in renin-induced hypertension remains unclear. Here the authors show that impaired vitamin D signaling in myeloid cells causes hypertension via macrophage-specific miR-106b-5p secretion, which activates renin production in the kidney.

    • J. Oh
    • S. J. Matkovich
    • C. Bernal-Mizrachi
    ResearchOpen Access
    Nature Communications
    Volume: 11, P: 1-12

  • Genetic and genomic approaches — including high-throughput sequence analysis and transcriptomics experiments — are revealing a clearer picture of the pathophysiological steps underlying the different forms of heart failure (genetic and acquired) and the genomic responses to cardiac overload.

    • Esther E. Creemers
    • Arthur A. Wilde
    • Yigal M. Pinto
    Reviews
    Nature Reviews Genetics
    Volume: 12, P: 357-362

  • This Review summarizes the latest findings on heart reverse remodelling, which demonstrate that despite apparent normalization of function, the molecular changes associated with heart failure persist in the reverse-remodelled heart. This myocardial remission is distinct from true recovery, in which both function and molecular makeup are normalized. These findings have implications for developing therapies to repair the failing heart.

    • Gene H. Kim
    • Nir Uriel
    • Daniel Burkhoff
    Reviews
    Nature Reviews Cardiology
    Volume: 15, P: 83-96