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Serological Changes Concomitant with the Growth of Mouse Tumours in Pretreated Rats

M. PIKOVSKI
C. TAL
E. MARGOLIASH
Research27 Jul 1957
Nature

Volume: 180, P: 185-186
CAR-engineered lymphocyte persistence is governed by a FAS ligand–FAS autoregulatory circuit

Klebanoff and colleagues report that survival and persistence of CAR-T and CAR-NK cells are regulated by a FAS ligand–FAS autoregulatory circuit, showing that disabling FAS signaling enhances their antitumor efficacy in preclinical models.

Fei Yi
Tal Cohen
Christopher A. Klebanoff
ResearchOpen Access22 Jul 2025
Nature Cancer

Volume: 6, P: 1638-1655
Ultrasensitive high-resolution profiling of early seroconversion in patients with COVID-19

A multiplexed fluorescence-based assay detects seroconversion in individuals infected with SARS-CoV-2 from less than 1 µl of blood as early as the day of the first positive nasopharyngeal nucleic acid test after symptom onset.

Maia Norman
Tal Gilboa
David R. Walt
Research18 Sept 2020
Nature Biomedical Engineering

Volume: 4, P: 1180-1187
A novel Fer/FerT targeting compound selectively evokes metabolic stress and necrotic death in malignant cells

The tyrosine-kinases Fer/FerT associate with the mitochondrial electron transport chain in cancer cells supporting their metabolic reprogramming. Here the authors discover a compound that disrupts Fer /FerT activity and selectively induces cell death of cancer cell lines displaying anti-tumor activity in vivo.

Yoav Elkis
Moshe Cohen
Uri Nir
ResearchOpen Access16 Oct 2017
Nature Communications

Volume: 8, P: 1-17
AUTS2 isoforms control neuronal differentiation

Galya Monderer-Rothkoff
Nitzan Tal
Sagiv Shifman
Research05 Apr 2019
Molecular Psychiatry

Volume: 26, P: 666-681

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Serological Changes Concomitant with the Growth of Mouse Tumours in Pretreated Rats

CAR-engineered lymphocyte persistence is governed by a FAS ligand–FAS autoregulatory circuit

Ultrasensitive high-resolution profiling of early seroconversion in patients with COVID-19

A novel Fer/FerT targeting compound selectively evokes metabolic stress and necrotic death in malignant cells

AUTS2 isoforms control neuronal differentiation

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