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Cardiac troponin I directly binds and inhibits mitochondrial ATP synthase with a noncanonical role in the post-ischemic heart

Elezaby et al. show that cardiac troponin I interacts with mitochondrial ATP synthase to increase ATPase activity. Disrupting this interaction reduces cardiac damage following transient ischemia.

Aly Elezaby
Amanda J. Lin
Daria Mochly-Rosen
Research18 Jul 2024
Nature Cardiovascular Research

Volume: 3, P: 987-1002
OPA1 helical structures give perspective to mitochondrial dysfunction

Cryo-electron microscopy structures of OPA1, mutations of which are associated with the disease dominant optic atrophy, provide insight into how structural features of OPA1 enable this protein to mediate mitochondrial-membrane fusion and remodelling.

Sarah B. Nyenhuis
Xufeng Wu
Jenny E. Hinshaw
Research23 Aug 2023
Nature

Volume: 620, P: 1109-1116
Host heparan sulfate promotes ACE2 super-cluster assembly and enhances SARS-CoV-2-associated syncytium formation

The molecular mechanism of syncytium formation during SARS-CoV-2 infection is not fully understood. Zhang et al. now show that cell surface heparan sulfate enhances spike-induced ACE2 clustering and cell-cell fusion, which depends on a conserved ACE2 linker and is blocked by a heparan sulfate binding drug.

Qi Zhang
Weichun Tang
Yihong Ye
ResearchOpen Access18 Sept 2023
Nature Communications

Volume: 14, P: 1-17

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