Author Correction: MicroRNA-302a promotes neointimal formation following carotid artery injury in mice by targeting PHLPP2 thus increasing Akt signaling

Correction to: Acta Pharmacologica Sinica https://doi.org/10.1038/s41401-020-0440-4, published online 21 July 2020

The authors apologized that the representative images of scratch assay and Transwell analysis (Fig. 5f, g) appeared incorrectly. The correct images are presented. The authors declare that these corrections do not change the results or conclusions of this paper. The authors apologize for any inconvenience caused to the journal and readers.

Fig. 5: Restoration of PHLPP2 or PTEN expression attenuates the effects of miR-302a on Akt activation, MASMC proliferation, and migration.

a–c The cells were cotransfected with miR-302a mimics and pCMV vector (vehicle), PTEN plasmid, or PHLPP2 plasmid for 24 h and then treated with PDGF-BB for another 1 h. Representative Western blotting images for p-Akt and Akt are shown (a). The ratio of p-Akt to Akt (b) and Akt activity (c) were measured. Cell viability (d) and cell cycle (e) analyses after PDGF-BB treatment for 24 h. Cell migratory capacity was examined by the scratch assay (f) and Transwell analysis (g). Scale bar, 200 μm for the scratch assay and 100 μm for the Transwell analysis. *P < 0.05 vs. PDGF-BB, ^#P < 0.05 vs^. PDGF-BB + 302a-m, n = 6.