Fig. 9

Galangin 3-methyl ether (G3-ME) alleviates pathological cardiac hypertrophy both in vivo and in vitro by targeting HDAC2 and subsequently deactivating the PI3K-AKT signaling pathway.

Galangin 3-methyl ether (G3-ME) alleviates pathological cardiac hypertrophy both in vivo and in vitro by targeting HDAC2 and subsequently deactivating the PI3K-AKT signaling pathway.