Fig. 5
HisCl1 in Rh6-expressing photoreceptors receive inputs both from outer and inner photoreceptors. a–c Actograms and phase plots. The experimental design is as described in Fig. 1 legend. a The RD phase advance rescue of CHO flies by HisCl1 expression in Rh6 photoreceptors (left) is slowed down by the absence of Rh1 (ninaE17 mutation, center) and is abolished by the absence of both Rh1 and Rh6 (ninaE17 Rh61 genetic background, right). b Flies lacking Cry, compound eyes, and ocelli but retaining the H-B eyelet synchronize with advanced LD cycles (so1; cry02 double mutants, left), but do not if HisCl1 expression is restricted to Rh6 photoreceptors (center). CHO mutants with HisCl1 rescue in Rh6 cells and genetic ablation of the eyelet (in addition to Rh5-expressing photoreceptors) synchronize with advanced RD cycles (right). c The LD phase advance rescue of CHO flies by HisCl1 expression in Rh6 photoreceptors is retained in the absence of either Rh1 and Rh6 (top) or Rh1, Rh5, and Rh6 (bottom)
