Fig. 1: A. thaliana aliphatic ITC- and GL-deficient mutants are hyper-susceptible to S. sclerotiorum. | Nature Communications

Fig. 1: A. thaliana aliphatic ITC- and GL-deficient mutants are hyper-susceptible to S. sclerotiorum.

From: The phytopathogenic fungus Sclerotinia sclerotiorum detoxifies plant glucosinolate hydrolysis products via an isothiocyanate hydrolase

Fig. 1: A. thaliana aliphatic ITC- and GL-deficient mutants are hyper-susceptible to S. sclerotiorum.The alt text for this image may have been generated using AI.

a Comparison of lesion areas caused by S. sclerotiorum 24 h post inoculation (hpi) on leaves of A. thaliana Col-0 wild-type, tgg1/tgg2 and myb28/myb29 mutants. The tgg1/tgg2 line is a myrosinase-defective mutant, and the myb28/myb29 line is deficient in aliphatic GL biosynthesis. b Relative quantification of S. sclerotiorum growth on A. thaliana lines for 24 h as quantified by qRT-PCR. The S. sclerotiorum Histone mRNA was normalized by the A. thaliana ACTIN2 mRNA to quantify relative fungal colonization. Data represent mean ± SEM (n = 6 inoculated leaves and plants, respectively; inoculation with detached leaves was repeated once) and were analyzed by one-way ANOVA (p < 0.001) followed by Tukey’s post-hoc test. Different letters above the bars indicate significant differences at p < 0.05. Source data are provided as a Source data file.

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