Fig. 3: Grx1 deficiency in chow-fed aged female Grx1-/- mice promotes atherosclerotic lesion formation. | Nature Communications

Fig. 3: Grx1 deficiency in chow-fed aged female Grx1-/- mice promotes atherosclerotic lesion formation.

From: Glutaredoxin 1 controls monocyte reprogramming during nutrient stress and protects mice against obesity and atherosclerosis in a sex-specific manner

Fig. 3

a, b Images of Oil Red O-stained aorta from male and female 18-month-old Grx1-/- Mice and age-matched male and female C57BL/6 J mice fed a normal chow. c Quantitation of aortic lesion area. n = 5–6 mice per group. d Distribution of lesions between aortic arch, descending aorta and abdominal aorta in female Grx1-/- mice, n = 5 mice per group. All data are expressed as mean ± S.D. One-way ANOVA followed by Fisher’s Least Significance Difference test was used to compare the mean values between experimental groups. Source data are provided as a Source Data file.

Back to article page