Fig. 7: Graphical abstract illustrating SYNCRIP controls proteome quality and CDC42-mediated cell polarity and division to maintain self-renewal of reserve HSCs. | Nature Communications

Fig. 7: Graphical abstract illustrating SYNCRIP controls proteome quality and CDC42-mediated cell polarity and division to maintain self-renewal of reserve HSCs.

From: RNA binding protein SYNCRIP maintains proteostasis and self-renewal of hematopoietic stem and progenitor cells

Fig. 7

Model showing mechanism for how SYNCRIP regulates HSPC properties. Left panel shows normal conditions with SYNCRIP and right panel shows HSPC conditions without SYNCRIP. Figure created with BioRender.com.

Back to article page