Fig. 6: A proposed working model for PINNA1-MtKNOX7 complex and MtUFO/MtKNOX7 module action in the compound leaf development in M. truncatula. | Nature Communications

Fig. 6: A proposed working model for PINNA1-MtKNOX7 complex and MtUFO/MtKNOX7 module action in the compound leaf development in M. truncatula.

From: Rewiring of a KNOXI regulatory network mediated by UFO underlies the compound leaf development in Medicago truncatula

Fig. 6: A proposed working model for PINNA1-MtKNOX7 complex and MtUFO/MtKNOX7 module action in the compound leaf development in M. truncatula.

In the wild type, PINNA1 represses the expression of MtKNOX7. Moreover, PINNA1 forms heterodimers with MtKNOX7 protein in the cytoplasmic space and attenuates the shuttling of MtKNOX7 from the cytoplasm into the nucleus. In pinna1 mutant, the expression domain of MtKNOX7 is expanded to leaf primordia, and more nuclear-localized MtKNOX7 proteins activate the transcription of MtUFO, thereby promoting indeterminacy in developing leaves.

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