Table 1 Summary of salivary gland basal cell adenoma (BCA) and basal cell adenocarcinoma (BCAC) cases

From: Wnt/β-catenin activation by mutually exclusive FBXW11 and CTNNB1 hotspot mutations drives salivary basal cell adenoma

Tumour type

DNA samples

RNA samples

Sex

Age, yrs (mean)

Site (Number of tumours)

BCAC

11 T/N

10 T

5 F

6 M

30–74 (55.2)

37–66 (55.4) F

30–74 (55.2) M

Parotid gland (7 primary, 1 recurrence)

Lung (1 metastasis, 1 salivary gland type carcinoma)

Sinonasal (1)

BCA

32 T/N

32 T

15 F

17 M

34–83 (60.1)

34–71 (53.9) F

36–84 (65.7) M

Parotid gland (29)

Submandibular gland (2)

Cheek (1)

  1. Number of tumour DNA samples with a matched normal sample, number of RNA samples, age and sex of patients in the BCA and BCAC cohorts used in this study. The BCAC cohort included 2 BCAC with differential diagnosis of epithelial-myoepithelial carcinoma. T tumour; N normal; F female; M male. Tumours are primary tumours unless otherwise indicated.
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