Table 3 Infectious complications in participating patients with head and neck carcinoma (HNC)

From: G-CSF/NAMPT signaling drives neutrophil dysfunction and enhances bacterial infection susceptibility in cancer patients

Group

Mean age (min-max) at admission, years

Mean time interval (min-max), months

Source of infection

Infectious agent

Gram-neg bacteria at admission

No infection (n = 10)

62.2 (49–76)

24.5 (7–36)

-

-

60% (no), 0% (yes), 40% (NA)

Later infection (n = 8)

54.7 (40–77)

4 (1–12)

See below

See below

12.5% (no), 62.5% (yes), 25% (NA)

ID19

  

Tracheal secret, bronchial secret

E. cloacae

Yes

ID25

  

Throat

E. coli

Yes

ID26

  

Breast

A. lwoffi

Yes

ID30

  

Blood (pneumogenic)

NA

No

ID32

  

Blood

B. cereus, S. oralis, E. coli, K. pneumoniae

Yes

ID40

  

Odontogenic

NA

NA

ID42

  

Pneumonia

E. coli

NA

ID50

  

Demerskatheter, BAL

S. epidermidis, Candida

Yes

  1. Baseline characteristics, infection timing, source, pathogenic agents, and Gram‑negative bacterial status at admission for patients with and without infectious complications during follow‑up. Mean age (range) at hospital admission, mean time interval (range) in months from diagnosis to infection, and detailed infection source and causative agents are shown where available. Gram‑negative bacterial status at admission is expressed as the percentage of patients without Gram‑negative bacteria (no), with Gram‑negative bacteria (yes), or with data not available (NA).
  2. HNC head and neck carcinoma, NA data not available, BAL bronchoalveolar lavage, E. cloacae Enterobacter cloacae, E. coli Escherichia coli, A. lwoffi Acinetobacter lwoffii, B. cereus Bacillus cereus, S. oralis Streptococcus oralis, K. pneumoniae Klebsiella pneumoniae, S. epidermidis Staphylococcus epidermidis.
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