Fig. 1: Immune fingerprints across respiratory viruses. | npj Digital Medicine

Fig. 1: Immune fingerprints across respiratory viruses.

From: Explainable AI multiomics analysis reveals shared and divergent host responses in COVID-19 and influenza

Fig. 1: Immune fingerprints across respiratory viruses.

Radar plot summarizing pathway-level activation for COVID-19, influenza, and RSV across six modules: type I/III interferon (IFN), IL-6/inflammation, T-cell activation, B-cell/antibody responses, neutrophil/NETosis, and coagulation/endothelium. Scores are normalized to 0-1, with larger radii indicating stronger activation. Profiles reveal a shared interferon signature with virus-specific bias–COVID-19 skewed toward neutrophil/NETosis and endothelial/coagulation axes, influenza toward adaptive (T/B-cell) responses, and RSV intermediate.

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