This article reports the case of a 49-year-old woman who presented to hospital with symptoms associated with severe hyponatremia. For the previous 6 years, she had been treated for lumbago with lumbar facet joint injections of triamcinolone acetonide every 10–12 weeks. Laboratory analyses and nonresponse to the corticotropin-releasing-hormone-stimulation test led to the conclusion that triamcinolone acetonide treatment had caused the development of secondary adrenal insufficiency, which in turn had led to upregulation of antidiuretic hormone and the development of hyponatremia.
- Stefan Reuter
- Niklas Scholten
- Eckhart Büssemaker
