Here the authors put forward a model in which the CtIP homolog Sae2 controls Mre11 endonuclease and 3’−5’ exonucleases via Rad50 by different mechanisms and reveals that the Mre11 endo-to-exonuclease switch is not required to process all blocked DNA ends
- Tomoki Tamai
- Giordano Reginato
- Katsunori Sugimoto
