Kathryn Anderson and colleagues use mouse genetics to define unexpected relationships between the retrograde ciliary motor gene Dync2h1 and other genes required for trafficking in primary cilia. Their findings argue that the mutant phenotypes in these models result from defects in cilia architecture rather than direct roles in signaling.
- Polloneal Jymmiel R Ocbina
- Jonathan T Eggenschwiler
- Kathryn V Anderson
