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Advanced filters: Author: Ourania Preza Clear advanced filters

  • Re-replication is a driving force of tumorigenesis and genomic instability. Here, the authors show that upon re-replication, FANCD2 localizes at early origins to limit replisome progression, ssDNA gap accumulation and fork breakage, revealing a vulnerability for selective targeting of cancer cells.

    • Nibal Badra-Fajardo
    • Elena Karydi
    • Zoi Lygerou
    ResearchOpen Access
    Nature Communications
    P: 1-22