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BCL11A deficiency protects epidermis from UVB-induced damage through promotion of autophagy

The deficiency of the essential transcriptional repressor BCL11A promotes cell autophagy to enhance the resistance of the epidermis to UVB through targeting the lipid synthesis gene ACSS3, rather than autophagy-related genes.

Xuyi Deng
Hui Liu
Meijuan Zhou
ResearchOpen Access02 Oct 2025
Communications Biology

Volume: 8, P: 1-9

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BCL11A deficiency protects epidermis from UVB-induced damage through promotion of autophagy

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