Nature Search

Author Correction: Mycobacterium tuberculosis-derived linoleic acid increases regulatory T cell function to promote bacterial survival within macrophages

Hongyu Cheng
Shenzhi Li
Baoxue Ge
Amendments and Corrections01 Dec 2025
Nature Microbiology

Volume: 11, P: 336
Mycobacterium tuberculosis-derived linoleic acid increases regulatory T cell function to promote bacterial survival within macrophages

Mycobacterium tuberculosis produces linoleic acid, which is released from macrophages during infection and upregulates regulatory T cell activity, to suppress macrophage control of intracellular bacteria in mice.

Hongyu Cheng
Shenzhi Li
Baoxue Ge
Research10 Oct 2025
Nature Microbiology

Volume: 10, P: 2949-2965
RIOK2 kinase regulates the translocation of the FADD–RIPK1–Caspase-8 complex to the ER and the cleavage of Gasdermin D to drive pyroptosis

Macrophage infection by Yersinia or specific stimulations trigger caspase-8-mediated GSDMD cleavage. RIOK2 kinase is essential for this process by interacting with FADD and driving the lysosome-to-ER transport of the cell death complex accompanied by GSDMD cleavage, thereby enhancing host defense.

Mingtong Ma
Fei Wang
Baoxue Ge
ResearchOpen Access17 Nov 2025
Nature Communications

Volume: 16, P: 1-17
Mycobacterium tuberculosis suppresses host antimicrobial peptides by dehydrogenating L-alanine

In this work, authors mechanistically investigate the reduced induction of antimicrobial peptides in Mycobacterium tuberculosis infected macrophages.

Cheng Peng
Yuanna Cheng
Lin Wang
ResearchOpen Access17 May 2024
Nature Communications

Volume: 15, P: 1-19
Mycobacterium tuberculosis produces d-serine under hypoxia to limit CD8⁺ T cell-dependent immunity in mice

Hypoxia induces d-serine production by Mycobacterium tuberculosis which inhibits mTORC1 and T-bet expression to restrict downstream CD8⁺ T cell immune responses and macrophage activation, and inhibit control of tuberculosis in mice.

Hongyu Cheng
Zhe Ji
Baoxue Ge
ResearchOpen Access28 May 2024
Nature Microbiology

Volume: 9, P: 1856-1872
Mycobacterium tuberculosis inhibits METTL14-mediated m⁶A methylation of Nox2 mRNA and suppresses anti-TB immunity

Mingtong Ma
Yongjia Duan
Baoxue Ge
ResearchOpen Access29 Mar 2024
Cell Discovery

Volume: 10, P: 1-23

Quick links

Search

Author Correction: Mycobacterium tuberculosis-derived linoleic acid increases regulatory T cell function to promote bacterial survival within macrophages

Mycobacterium tuberculosis-derived linoleic acid increases regulatory T cell function to promote bacterial survival within macrophages

RIOK2 kinase regulates the translocation of the FADD–RIPK1–Caspase-8 complex to the ER and the cleavage of Gasdermin D to drive pyroptosis

Mycobacterium tuberculosis suppresses host antimicrobial peptides by dehydrogenating L-alanine

Mycobacterium tuberculosis produces d-serine under hypoxia to limit CD8⁺ T cell-dependent immunity in mice

Mycobacterium tuberculosis inhibits METTL14-mediated m⁶A methylation of Nox2 mRNA and suppresses anti-TB immunity

Search

Quick links

Search

Filter By:

Author Correction: Mycobacterium tuberculosis-derived linoleic acid increases regulatory T cell function to promote bacterial survival within macrophages

Mycobacterium tuberculosis-derived linoleic acid increases regulatory T cell function to promote bacterial survival within macrophages

RIOK2 kinase regulates the translocation of the FADD–RIPK1–Caspase-8 complex to the ER and the cleavage of Gasdermin D to drive pyroptosis

Mycobacterium tuberculosis suppresses host antimicrobial peptides by dehydrogenating L-alanine

Mycobacterium tuberculosis produces d-serine under hypoxia to limit CD8+ T cell-dependent immunity in mice

Mycobacterium tuberculosis inhibits METTL14-mediated m6A methylation of Nox2 mRNA and suppresses anti-TB immunity

Search

Quick links

Mycobacterium tuberculosis produces d-serine under hypoxia to limit CD8⁺ T cell-dependent immunity in mice

Mycobacterium tuberculosis inhibits METTL14-mediated m⁶A methylation of Nox2 mRNA and suppresses anti-TB immunity