Fig. 5: Mutation of AKT1 reduces the salt tolerance of bzip49cr mutant.
From: SC35-mediated bZIP49 splicing regulates K⁺ channel AKT1 for salt stress adaptation in poplar
a Phenotypes comparison of 60-day-old WT, akt1cr, bzip49cr and akt1cr/bzip49cr mutant plants under control and 100 mM NaCl conditions. b–e Measurements of chlorophyll content (b), Fv/Fm (c), K+ content (d), and Na+ content (e) of WT, akt1cr, bzip49cr, and akt1cr/bzip49cr mutant plants under normal and salt stress conditions. Box plots show medians, interquartile ranges, and 1.5×IQR whiskers; dots indicate outliers (n = 12 biological replicates). f–i Net K+ (f, h) and Na+ (g, i) fluxes in the root meristem and elongation zone measured using NMT. Line and bar charts display mean values across the time course and replicates. Values are means ± SE (n = 3 biological replicates). Different letters indicate significant differences (P < 0.05, two-way ANOVA; P values are shown in Source Data file). Source data are provided as a Source Data file.
